epiderm™ normal human skin substitutes Search Results


epiderm™ normal human skin substitutes  (MatTek)
90
MatTek epiderm™ normal human skin substitutes
Epiderm™ Normal Human Skin Substitutes, supplied by MatTek, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/epiderm%E2%84%A2+normal+human+skin+substitutes/epiderm++normal+human+skin+substitutes/us10195245-118-31-38
Average 90 stars, based on 1 article reviews
epiderm™ normal human skin substitutes - by Bioz Stars, 2026-10
90/100 stars
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reconstructed normal human three-dimensional skin epiderm  (MatTek)
90
MatTek reconstructed normal human three-dimensional skin epiderm
Immunohistochemical analysis of cyclobutane pyrimidine dimers in nasal epithelial tissues from patients subsequent to treatment with rhinophototherapy. Monoclonal antibodies specific for CPDs were used to visualize DNA damage in the nasal <t>epithelium</t> of patients 2 months after the final exposure to a RPT treatment for seasonal allergies ( i.e . 9 treatments over a 3-week period). Tissue sections are shown for RPT-treated patients (C), sham-treated (visible light only) patients (B) and a positive control of UV irradiated 3D reconstructed normal human <t>respiratory</t> epithelium <t>(EpiAirway)</t> (A).
Reconstructed Normal Human Three Dimensional Skin Epiderm, supplied by MatTek, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/epiderm%E2%84%A2+normal+human+skin+substitutes/reconstructed+normal+human+three+dimensional+skin+epiderm/pmc03837592-36-30-36
Average 90 stars, based on 1 article reviews
reconstructed normal human three-dimensional skin epiderm - by Bioz Stars, 2026-10
90/100 stars
  Buy from Supplier

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Immunohistochemical analysis of cyclobutane pyrimidine dimers in nasal epithelial tissues from patients subsequent to treatment with rhinophototherapy. Monoclonal antibodies specific for CPDs were used to visualize DNA damage in the nasal epithelium of patients 2 months after the final exposure to a RPT treatment for seasonal allergies ( i.e . 9 treatments over a 3-week period). Tissue sections are shown for RPT-treated patients (C), sham-treated (visible light only) patients (B) and a positive control of UV irradiated 3D reconstructed normal human respiratory epithelium (EpiAirway) (A).

Journal: Journal of Cellular and Molecular Medicine

Article Title: Molecular response of nasal mucosa to therapeutic exposure to broad-band ultraviolet radiation

doi: 10.1111/j.1582-4934.2008.00442.x

Figure Lengend Snippet: Immunohistochemical analysis of cyclobutane pyrimidine dimers in nasal epithelial tissues from patients subsequent to treatment with rhinophototherapy. Monoclonal antibodies specific for CPDs were used to visualize DNA damage in the nasal epithelium of patients 2 months after the final exposure to a RPT treatment for seasonal allergies ( i.e . 9 treatments over a 3-week period). Tissue sections are shown for RPT-treated patients (C), sham-treated (visible light only) patients (B) and a positive control of UV irradiated 3D reconstructed normal human respiratory epithelium (EpiAirway) (A).

Article Snippet: In addition, we quantified and compared the induction and repair of the two major photoproducts formed after exposure to UV, the CPD and (6–4) photoproduct, in reconstructed normal human three-dimensional respiratory epithelium (EpiAirway) and skin (EpiDerm) (MatTek Corp, Ashland, MA, USA).

Techniques: Immunohistochemical staining, Bioprocessing, Positive Control, Irradiation

DNA damage distribution in 3D reconstructed normal human respiratory epithelia (EpiAirway) exposed to different light sources. Immunohistochemistry using monoclonal antibodies specific for CPDs is shown immediately after irradiation with a broad-band UVC/UVB lamp used for RPT (Allux Medical) (C), a narrow-band Philips TL01 UVB lamp (B), a narrow-band UVC germicidal lamp (A) and the broadband UVC/UVB lamp shown in Panel A from which the UVB component had been removed (D).

Journal: Journal of Cellular and Molecular Medicine

Article Title: Molecular response of nasal mucosa to therapeutic exposure to broad-band ultraviolet radiation

doi: 10.1111/j.1582-4934.2008.00442.x

Figure Lengend Snippet: DNA damage distribution in 3D reconstructed normal human respiratory epithelia (EpiAirway) exposed to different light sources. Immunohistochemistry using monoclonal antibodies specific for CPDs is shown immediately after irradiation with a broad-band UVC/UVB lamp used for RPT (Allux Medical) (C), a narrow-band Philips TL01 UVB lamp (B), a narrow-band UVC germicidal lamp (A) and the broadband UVC/UVB lamp shown in Panel A from which the UVB component had been removed (D).

Article Snippet: In addition, we quantified and compared the induction and repair of the two major photoproducts formed after exposure to UV, the CPD and (6–4) photoproduct, in reconstructed normal human three-dimensional respiratory epithelium (EpiAirway) and skin (EpiDerm) (MatTek Corp, Ashland, MA, USA).

Techniques: Immunohistochemistry, Bioprocessing, Irradiation

Nucleotide excision repair kinetics in 3D reconstructed normal human nasal epithelial and epidermal tissues. CPD and (6–4)PD repair are shown in EpiDerm (•–•) and EpiAirway (○–○) artificial tissues. The upper panels show the frequencies of CPDs (A) and (6–4)PDs (B) at increasing times post-irradiation as lesions per megabase DNA. Directly below each panel the frequencies have been normalized to the amount of damage measured at T 0 and are expressed as the percentage of CPDs (C) and (6–4)PDs (D) remaining at increasing times post-irradiation with a single sublethal dose of UVB. Exponential decay curves for each data set are shown. Standard errors of the mean were calculated from standard deviations using a total of 8 data points from duplicate tissues.

Journal: Journal of Cellular and Molecular Medicine

Article Title: Molecular response of nasal mucosa to therapeutic exposure to broad-band ultraviolet radiation

doi: 10.1111/j.1582-4934.2008.00442.x

Figure Lengend Snippet: Nucleotide excision repair kinetics in 3D reconstructed normal human nasal epithelial and epidermal tissues. CPD and (6–4)PD repair are shown in EpiDerm (•–•) and EpiAirway (○–○) artificial tissues. The upper panels show the frequencies of CPDs (A) and (6–4)PDs (B) at increasing times post-irradiation as lesions per megabase DNA. Directly below each panel the frequencies have been normalized to the amount of damage measured at T 0 and are expressed as the percentage of CPDs (C) and (6–4)PDs (D) remaining at increasing times post-irradiation with a single sublethal dose of UVB. Exponential decay curves for each data set are shown. Standard errors of the mean were calculated from standard deviations using a total of 8 data points from duplicate tissues.

Article Snippet: In addition, we quantified and compared the induction and repair of the two major photoproducts formed after exposure to UV, the CPD and (6–4) photoproduct, in reconstructed normal human three-dimensional respiratory epithelium (EpiAirway) and skin (EpiDerm) (MatTek Corp, Ashland, MA, USA).

Techniques: Irradiation